Mol Microbiol 2003,50(1):101–104 PubMedCrossRef 63 Boles BR, Hor

Mol Microbiol 2003,50(1):101–104.PubMedCrossRef 63. Boles BR, Horswill AR: Agr-mediated dispersal of Staphylococcus aureus biofilms. learn more PLoS Pathog 2008,4(4):e1000052.PubMedCrossRef 64. Lepine F, Milot S, Deziel E, He JX, Rahme LG: Electrospray/mass spectrometric identification and analysis of 4-hydroxy-2-alkylquinolines (HAQs) produced by Pseudomonas aeruginosa . J Am Soc

Mass Spectr 2004,15(6):862–869.CrossRef 65. Haussler S, Becker T: The Pseudomonas quinolone signal (PQS) balances life and death in Pseudomonas aeruginosa populations. PLoS Pathog 2008,4(9):e1000166.PubMedCrossRef 66. Mashburn-Warren L, Howe J, Garidel P, Richter W, Steiniger F, Roessle M, Brandenburg K, Whiteley M: Interaction of quorum signals with outer membrane lipids: insights into prokaryotic membrane vesicle formation.

Mol Microbiol 2008,69(2):491–502.PubMedCrossRef 67. Ventre I, Goodman AL, Vallet-Gely I, Vasseur P, Soscia C, Molin S, Bleves S, Lazdunski A, Lory S, Emricasan clinical trial Filloux A: Multiple sensors control reciprocal expression of Pseudomonas aeruginosa regulatory RNA and virulence genes. Proc Natl Acad Sci USA 2006,103(1):171–176.PubMedCrossRef 68. Brencic A, Lory S: Determination of the regulon and identification of novel mRNA targets of Pseudomonas aeruginosa RsmA. Mol Microbiol 2009,72(2):612–632.PubMedCrossRef Authors’ contributions MS and RG designed and RG performed the experiments. RG and MS analyzed and interpreted the results. RG drafted

the manuscript and MS critically revised it. All authors read and approved the final manuscript.”
“Background Citrus canker, caused by the Gram-negative plant pathogenic bacterium Xanthomonas citri subsp. citri (Xac) (syn. Xanthomonas axonopodis pv. citri) [1, 2], is one of the most important diseases of citrus crop worldwide [3]. Citrus canker is widely distributed in wet subtropical citrus growing areas and affects most commercial citrus selleck inhibitor varieties [3, 4]. The canker symptom is characterized by raised necrotic lesions on leaves, stems and fruit of infected trees; and in severe cases, defoliation, twig dieback, general tree decline, blemished fruit and premature fruit drop can occur Dolichyl-phosphate-mannose-protein mannosyltransferase [3, 4]. Wind-blown rain is the primary short- to medium-distance spread mechanism for citrus canker and long-distance dissemination is usually caused by transportation of infected citrus fruits and plant materials [5]. The decrease of yield and less value or entirely unmarketable of infected fruit are responsible for serious economic losses [3]. Moreover, this disease has a significant impact on commerce due to restrictions to national and international fruit trade from canker-affected areas [3]. Economic losses are also resulting from costly eradication programs and heave use of chemical treatments such as copper-based bactericides for prevention from and control of citrus canker disease [6].

Comments are closed.